A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Mutation in blood coagulation factor V associated with resistance to activated protein C. Blood, 87pp.
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Protein C and S deficiency, thrombofilia and hypofibrinolysis: Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease. Lancet,pp.
Familial hypofibrinolysis and venous thrombosis. Blood, 82pp. Br J Hematol, 71pp. J Med,pp. J Biol Chem,pp. Non traumatic osteonecrosis of the femoral head: You can change the settings or obtain more information by clicking here.
Enfermedad de Legg-Calvé-Perthes – Síntomas y causas – Mayo Clinic
Disorders of hemostasis in childhood: Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
The remaining patients were considered withinthe normal range when age was taken into account. The Kinston antiphospholipid group. Thromb Res, 11pp. Familial idiopathic oeteonecrosis mediated by familial hypofibrinolysis witn high levels of plasminogen activator inhibitor.
¿Trombofilia y enfermedad de Perthes? | Anales de Pediatría
The second international anticardiolipin standardization workshop. Enfermrdad Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Clin Orthop,pp. Thromb Haemost, 71pp.
Anticoagulant protein C pathway defective in majority of thromboembolic patients. Decreased petthes potential in patients with idio-pathic avascular necrosis and transient osteoporosis of the hip.
Severe thrombotic disease in a young man with bone marrow and skeletal changes and with a high content of an inhibitor in the fibrinolytic system.
Perthes’ disease and the relevance of thrombophilia. Blood collection pediatrai strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity. Pediatr Res, 35pp.
Variability of thrombosis among homozigous siblings with resistance to activated protein C due to an Arg-Gln mutaion in gen for factor V. Clin Chem, 32pp.
Pathophysiology of osteonecrosis of the jaw: J Clin Invest, 94pp. Are you a health professional able to prescribe or dispense drugs?
Síndrome de Legg-Calvé-Perthes
Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families. Changes in coagulation-fibrinolysis system in Legg-Perthes disease: Espectrophotometric solid-phase tissue plasminogen ee activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators.
Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.